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Cold environment exacerbates brain pathology and oxidative stress following traumatic brain injuries: Potential therapeutic effects of nanowired antioxidant compound H-290/51
Blood-brain barrier
Brain edema
Cold environment
Glutathione
H-290/51
Lucigenin
Luminol
Malondialdehyde
Nanodelivery
Neuronal damage
Oxidative stress
Traumatic brain injur...
antioxidant
glutathione
H 290 51
h 290 51
lucigenin
luminol
malonaldehyde
nanowire
titanium dioxide
unclassified drug
antioxidant
H290-51
indole derivative
nanowire
neuroprotective agent...
animal experiment
animal model
animal tissue
Article
blood brain barrier
brain region
cold acclimatization
controlled study
disease exacerbation
dose response
drug delivery system
drug effect
edema
enzyme activity
enzyme blood level
male
neuropathology
neuroprotection
nonhuman
oxidative stress
parietal lobe
pathophysiology
rat
room temperature
traumatic brain injur...
animal
brain
cold
metabolism
oxidative stress
pathology
physiology
Sprague Dawley rat
traumatic brain injur...
treatment outcome
Animals
Antioxidants
Brain
Brain Injuries, Traum...
Cold Temperature
Indoles
Male
Nanowires
Neuroprotective Agent...
Oxidative Stress
Rats
Rats, Sprague-Dawley
Treatment Outcome