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Non-cytotoxic copper overload boosts mitochondrial energy metabolism to modulate cell proliferation and differentiation in the human erythroleukemic cell line K562
Fecha de emisión
2016
Autor(es)
Ruiz L.M.
Jensen E.L.
Rossel Y.
Puas G.I.
Gonzalez-Ibanez A.M.
Bustos R.I.
Ferrick D.A.
Elorza A.A.
DOI
10.1016/j.mito.2016.04.005
Temas
Autophagy
Bioenergetics
Copper
Erythropoiesis
Mitochondria
Mitochondrial dynamic...
adenosine triphosphat...
copper
reactive oxygen metab...
copper
aerobic metabolism
Article
autophagy
bioenergy
cell differentiation
cell fate
cell function
cell proliferation
cell structure
controlled study
copper overload
energy metabolism
enzyme synthesis
erythroleukemia cell
human
human cell
mitochondrial dynamic...
mitochondrial membran...
mitochondrial respira...
nutritional disorder
oxygen consumption
priority journal
upregulation
drug effects
energy metabolism
K-562 cell line
metabolism
mitochondrion
Cell Differentiation
Cell Proliferation
Copper
Energy Metabolism
Humans
K562 Cells
Mitochondria
Archivo(s)