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Myofibers deficient in connexins 43 and 45 expression protect mice from skeletal muscle and systemic dysfunction promoted by a dysferlin mutation
Fecha de emisión
2020
Autor(es)
Fernández G.
Arias-Bravo G.
Bevilacqua J.A.
Castillo-Ruiz M.
Caviedes P.
Sáez J.C.
Cea L.A.
DOI
10.1016/j.bbadis.2020.165800
Temas
Calcium ion
Fat infiltration
Membrane permeability...
Muscular dystrophy
Muscular performance
connexin 39
connexin 43
connexin 45
creatine kinase
dysferlin
gap junction protein
myogenin
unclassified drug
adult
animal cell
animal experiment
animal model
animal tissue
Article
calcium cell level
calcium signaling
cell membrane permeab...
comparative study
connective tissue
controlled study
creatine kinase blood...
dysferlinopathy
enzyme activity
gastrocnemius muscle
gene mutation
human
immunoreactivity
lipid storage
male
molecular pathology
motor performance
mouse
muscle atrophy
muscle cell
muscle function
muscle injury
muscle tissue
nonhuman
priority journal
protein expression
rotarod test
sarcolemma
skeletal muscle
Archivo(s)