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dc.contributor.authorLaborde, Carlos M.
dc.contributor.authorLarzabal, Leyre
dc.contributor.authorGonzález-Cantero, Álvaro
dc.contributor.authorCastro-Santos, Patricia
dc.contributor.authorDíaz-Peña, Roberto
dc.date.accessioned2022-03-02T18:39:45Z
dc.date.available2022-03-02T18:39:45Z
dc.date.issued2022-01
dc.identifier10.3390/jpm12010035
dc.identifier.issn20754426
dc.identifier.urihttps://hdl.handle.net/20.500.12728/9952
dc.description.abstractPsoriatic arthritis (PsA) is a common type of inflammatory arthritis found in up to 40% of patients with psoriasis. Although early diagnosis is important for reducing the risk of irreversible structural damage, there are no adequate screening tools for this purpose, and there are no clear markers of predisposition to the disease. Much evidence indicates that PsA disorder is complex and heterogeneous, where genetic and environmental factors converge to trigger inflammatory events and the development of the disease. Nevertheless, the etiologic events that underlie PsA are complex and not completely understood. In this review, we describe the existing data in PsA in order to highlight the need for further research in this disease to progress in the knowledge of its pathobiology and to obtain early diagnosis tools for these patients.es_ES
dc.language.isoenes_ES
dc.publisherMDPIes_ES
dc.subjectEpigenomicses_ES
dc.subjectGenomewide association studieses_ES
dc.subjectGenomicses_ES
dc.subjectPersonalized medicinees_ES
dc.subjectPsoriatic arthritises_ES
dc.subjectSpondyloarthropathieses_ES
dc.subjectTranscriptomicses_ES
dc.titleAdvances of Genomic Medicine in Psoriatic Arthritises_ES
dc.typeArticlees_ES


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