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Pathogenic Mechanisms in Chronic Obstructive Pulmonary Disease Due to Biomass Smoke Exposure [Mecanismos patogénicos en la enfermedad pulmonar obstructiva crónica causada por exposición a humo de biomasa]

dc.contributor.authorSilva R.
dc.contributor.authorOyarzún M.
dc.contributor.authorOlloquequi J.
dc.date.accessioned2020-09-02T22:28:17Z
dc.date.available2020-09-02T22:28:17Z
dc.date.issued2015
dc.identifier10.1016/j.arbr.2015.04.013
dc.identifier.citation51, 6, 285-292
dc.identifier.issn03002896
dc.identifier.urihttps://hdl.handle.net/20.500.12728/6266
dc.descriptionChronic obstructive pulmonary disease (COPD) mortality and morbidity have increased significantly worldwide in recent decades. Although cigarette smoke is still considered the main risk factor for the development of the disease, estimates suggest that between 25% and 33% of COPD patients are non-smokers. Among the factors that may increase the risk of developing COPD, biomass smoke has been proposed as one of the most important, affecting especially women and children in developing countries.Despite the epidemiological evidence linking exposure to biomass smoke with adverse health effects, the specific cellular and molecular mechanisms by which this pollutant can be harmful for the respiratory and cardiovascular systems remain unclear. In this article we review the main pathogenic mechanisms proposed to date that make biomass smoke one of the major risk factors for COPD. © 2014 SEPAR.
dc.language.isoes
dc.publisherElsevier Doyma
dc.subjectBiomass smoke
dc.subjectChronic obstructive pulmonary disease
dc.subjectGenotoxicity
dc.subjectInflammation
dc.subjectPathogenesis
dc.subjectbiomass
dc.subjectbiomass smoke
dc.subjectchronic obstructive lung disease
dc.subjectdeveloping country
dc.subjectenvironmental exposure
dc.subjecthuman
dc.subjectmorbidity
dc.subjectmortality
dc.subjectpathogenesis
dc.subjectrisk factor
dc.subjectShort Survey
dc.subjectsmoke
dc.subjectsmoking
dc.subjectage
dc.subjecthigh risk population
dc.subjectinflammation
dc.subjectmolecular mechanics
dc.subjectoxidative stress
dc.subjectpathogenicity
dc.subjectpollutant
dc.subjectsex
dc.subjectadult
dc.subjectadverse effects
dc.subjectair pollutant
dc.subjectanimal
dc.subjectchild
dc.subjectcomplication
dc.subjectcooking
dc.subjectdrug effects
dc.subjectenvironmental exposure
dc.subjectfemale
dc.subjectgas
dc.subjectheating
dc.subjectindoor air pollution
dc.subjectinflammation
dc.subjectlung burn
dc.subjectmale
dc.subjectmetabolism
dc.subjectmouse
dc.subjectparticulate matter
dc.subjectpregnancy
dc.subjectprenatal exposure
dc.subjectPulmonary Disease, Chronic Obstructive
dc.subjectrural population
dc.subjectsecretion (process)
dc.subjectsmoke
dc.subjectascorbic acid
dc.subjectglutathione
dc.subjectinterleukin 6
dc.subjectinterleukin 8
dc.subjectreactive oxygen metabolite
dc.subjectsuperoxide dismutase
dc.subjecttobacco smoke
dc.subjecttumor necrosis factor
dc.subjectair pollutant
dc.subjectautacoid
dc.subjectcytokine
dc.subjectgas
dc.subjectinflammasome
dc.subjectparticulate matter
dc.subjectsmoke
dc.subjecttransient receptor potential channel
dc.subjectAdult
dc.subjectAir Pollutants
dc.subjectAir Pollution, Indoor
dc.subjectAnimals
dc.subjectBiomass
dc.subjectChild
dc.subjectCooking
dc.subjectCytokines
dc.subjectEnvironmental Exposure
dc.subjectFemale
dc.subjectGases
dc.subjectHeating
dc.subjectHumans
dc.subjectInflammasomes
dc.subjectInflammation
dc.subjectInflammation Mediators
dc.subjectMale
dc.subjectMice
dc.subjectOxidative Stress
dc.subjectParticulate Matter
dc.subjectPregnancy
dc.subjectPrenatal Exposure Delayed Effects
dc.subjectPulmonary Disease, Chronic Obstructive
dc.subjectRural Population
dc.subjectSmoke
dc.subjectSmoke Inhalation Injury
dc.subjectTransient Receptor Potential Channels
dc.titlePathogenic Mechanisms in Chronic Obstructive Pulmonary Disease Due to Biomass Smoke Exposure [Mecanismos patogénicos en la enfermedad pulmonar obstructiva crónica causada por exposición a humo de biomasa]
dc.typeShort Survey


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